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A fundamental challenge for every living cell is maintaining the integrity of its DNA. Mutations, double-strand breaks (DSBs), and other lesions occur constantly due to environmental factors and internal metabolic processes [9, 15]. To manage this, cells have evolved sophisticated [14].

By mapping which repair genes are "on" or "off" during specific phases of the cell cycle, scientists can identify:

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The research by Mjelle and Hegre utilizes advanced techniques like and pseudotime analysis to track how gene expression changes second-by-second as a cell matures [13, 21]. This level of detail helps explain how "The Guardian of the Genome" (the p53 protein) and other regulators keep us healthy [12]. If you'd like to explore this further, I can provide:

: Understanding when cells are most "fragile" allows for better timing of chemotherapy and radiotherapy, which work by inducing DNA damage [9, 22]. Scientific Impact By mapping which repair genes are "on" or

A summary of (like RAD51 or OGG1) mentioned in their papers [14, 23].